The
following article was written in response to an article claiming that the
cause of HD within our breed lay in diet.
Read
original article here
Cause of HD — A Summary
There will always be those who rebel against evidence,
and pick a seeming exception on which to build a contrarian theory. It’s
like the cults that take a verse of the Bible, misinterpret or mistranslate
it, and build a whole religion on it. They balance an Egyptian pyramid
upside-down on its pointed head. Eventually such a structure will fall.
The bane of the scientist
has always been the multitude of anecdotal reports that, like mushrooms,
pop up suddenly and unexpectedly in the dark. One such report (without
scientific controls) from Norway in the early 1990s argued that in five
breeds, but not two others, HD was more likely to show up in puppies that
grew up in the fall and winter. As in other amateur observations, questions
were raised regarding exercise and sunlight (vitamin D) among other differences.
In fact, most attempts to pin the blame of HD onto environment involve
diet and exercise. While we must not reject out of hand all anecdotal claims,
we must also place our bets on the repeatable, controlled studies, if we
want to gain true knowledge. People have blamed the brand of dog food,
the footing puppies were raised on, and other things for the appearance
of dysplasia in puppies that they have sold, with or without guarantees
and/or stipulations. There is no scientific basis for these excuses people
use in order to not make good on defective “products”.
On the other hand, there is plenty of support for
the fact that “overnutrition” and more rapid growth are responsible for
the genetically pre-disposed dog to develop worse and earlier HD than might
be the case if on a restricted diet. My new revision of the HD book will
deal with this in detail. While some have claimed that exercise is needed
to maintain a “deep acetabulum”, there is likewise no controlled study
to back up this concept. It is “common wisdom” that hip-extended radiographs
seem to show less laxity in young dogs that have been kept lean and frequently
exercised, such as in training for schutzhund and police work, than found
in dogs living a more relaxed life, but stress radiography shows every
case of laxity. Muscle tone can have some positive effect on the tightness
of the joint capsule, as long as regular exercise is maintained. It has
no effect, however, on the genes, so a dog that has been “lean and mean”
until its skeleton had fully ossified, is just as dangerous to breed to
as is his littermate who has reclined on a couch most of his growing-up
life.
GENES, NUTRITION, AND DENIAL
At the end of Year 2000,
an article appeared in the underground media, primarily Internet and e-mail
discussion groups, called “The Error of the Millennium in Veterinary Medicine”,
a sequel to a “compendium” pamphlet called “The Thirty Years’ War”. The
reason it was there but was refused by all legitimate journals and magazines
was that its premise was so preposterous. It reminded me of the inflammatory
claim of the same decade that the Nazi Holocaust never happened. Of course,
history is full of examples of error that promenade as fact, and discoveries
that are suppressed for long years until someone else proves their validity.
For one, an Australian researcher named Barry Marshall stated that stomach
ulcers were caused by bacteria, and even proved it by swallowing the microbes,
developing ulcers, and curing them with antibiotics. Yet the textbooks
for years continued to give the “risk factors” as smoking, alcohol, stress,
and genes. We now know that TB and peptic ulcers are infectious disorders,
as the November 27, 2000 issue of Newsweek stated, in its review of “Plague
Time”. In that new book, Amherst biologist Paul Ewald put forth the equally
revolutionary concept that parasitic germs are the cause of many forms
of Alzheimer’s, heart disease, cancer, mental illness, etc.
But the exception does
not become the rule. A lot of error can be cloaked in a very little truth,
and if viewers want to see only the flashy ornamentation, then that’s
all they will see. The authors of the current heresy that HD is
not genetic, but is caused by nutrition, make at least as many false claims
as did the purveyor of ascorbate as a preventative and cure for dysplasia.
While doing so, they hypothesize (or fabricate, really) a monstrous conspiracy
drama in which the villains are “the dog breeding associations in the United
States, United Kingdom, France, and Germany [that] developed X-ray systems”.
They claim that attempts to eliminate HD through selective breeding “proved
fruitless” for three decades, and they make the illogical conclusion that
this is the reason the incidence “persists at around 60-65%”. Quoting themselves
in their book, a vet named Torel and a journalist named Kammerer make amazing
far-out statements such as “breeding programs… cannot hope to bring about
any fundamental improvements …because CHD is not heritable and existing
dog food… is in fact the original cause of CHD.”
How ridiculous this is,
can quickly become clear by asking yourself why the dogs I and countless
others have bred selectively for good hips are not ruined
in spite of lavish helpings or restricted diets (doesn’t matter). Why do
Greyhounds not get HD, when they eat the same rations that Clumber Spaniels
do? Why are some of us who use selective breeding successful, while those
who do
not, have multiple failures, and yet we all feed basically
the same commercial preparations?
The conspiracy theory includes
accusations that the national all-breed club in Germany colluded with the
veterinary association and the Association of Publishers and Booksellers
to have the Torel-Kammerer book banned by the “biased and prejudicially
influenced” courts, and were supported by “behind-the-scenes manipulation
among the judges”. The VDH, the vet association, and leading publishers
are accused of “fraud, unfair competition (?) and corruption with active
and passive bribery.” The Zuchtwert (breed value) system headed by Prof.
Beuing is called “a piece of profitable charlatanism”. The diatribe that
goes on for pages would take too many additional pages to debunk, but the
work has more holes than a Swiss cheese factory.
SUMMARY
The base cause of HD and
other orthopedic diseases is a collection of enough genes that act together
to bring on the condition and the signs. Since it is extremely expensive
and difficult to find specific genes, even in this day of genome mapping,
it is more convenient, when speaking of the “causes”, etiology, or pathogenesis
(they all mean much the same thing), to focus on the secondary causes.
These may include development of a deep or shallow acetabulum, weak musculature
and ligaments, speed of growth of bones vs. muscles, synovial fluid aberrations,
and other features which might be identified early, but remember that all
these have their own “bad genes”. No matter how much research into the
role of those factors you support, you must still practice good genetics
if you wish to make progress in your own kennel or breed
Dr. Lust of Cornell, for
example, says flat out “Canine hip dysplasia is a hereditary disease”.
All of the best work of hundreds of dedicated scientists in a score of
countries is summed up in that simple, concise statement of truth. Unadorned,
forceful, and direct, this says it all.
Fred Lanting, author of the book on HD and worldwide
lecturer on the subject, is working on a greatly expanded version of the
book.
Fred Lanting's latest book:
“The Total German Shepherd Dog”
